Andreas Frick
Chef d'équipe
Directeur de recherche / Inserm/ Contact
+33 5 57 57 37 04
/ Adresse
Neurocentre Magendie - INSERM U 1215 - 146, rue Léo Saignat - 33077 Bordeaux
Publications
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Endogenous noise of neocortical neurons correlates with atypical sensory response variability in the Fmr1−/y mouse model of autism
Nat Commun. 2023-11-30. 14(1)
10.1038/s41467-023-43777-z -
Detecting fine and elaborate movements with piezo sensors provides non-invasive access to overlooked behavioral components
Neuropsychopharmacol.. 2021-11-11.
10.1038/s41386-021-01217-w -
Probing the polarity of spontaneous perisomatic GABAergic synaptic transmission in the mouse CA3 circuit in vivo.
Cell Reports. 2021-07-01. 36(2) : 109381.
10.1016/j.celrep.2021.109381 -
COUP-TFI/Nr2f1 Orchestrates Intrinsic Neuronal Activity during Development of the Somatosensory Cortex
Cerebral Cortex. 2020-06-23.
10.1093/cercor/bhaa137 -
Intrinsic neuronal excitability and spontaneous activity underlie cortical abnormalities upon Nr2f1/COUP-TFI deficiency
. 2019-08-08.
10.1101/728402 -
Probing the polarity of spontaneous GABAergic synaptic transmission in vivo
. 2019-05-07.
10.1101/630608 -
Hippocampal mossy fibers synapses in CA3 pyramidal cells are altered at an early stage in a mouse model of Alzheimer’s disease.
Journal of Neuroscience. 2019-03-18.
10.1523/JNEUROSCI.2868-18.2019 -
Altered surface mGluR5 dynamics provoke synaptic NMDAR dysfunction and cognitive defects in Fmr1 knockout mice
Nat Commun. 2017-10-24. 8(1)
10.1038/s41467-017-01191-2 -
Behavioral abnormalities in the Fmr1-KO2 mouse model of fragile X syndrome: The relevance of early life phases
Autism Research. 2017-06-07. 10(10) : 1584-1596.
10.1002/aur.1814 -
Chronic cannabinoid receptor stimulation selectively prevents motor impairments in a mouse model of Huntington’s disease.
Neuropharmacology. 2015-02-01. 89 : 368-374.
10.1016/j.neuropharm.2014.07.021 -
Dendritic channelopathies contribute to neocortical and sensory hyperexcitability in Fmr1(-/y) mice.
Nat Neurosci. 2014-11-10. 17(12) : 1701-1709.
10.1038/nn.3864